A cure for Tuberculosis

Vinegar has been used for thousands of years as a drug resistant common disinfectant. It shows positive result for mitigating tuberculosis. Clinical trials were carried mycobacteriology labs for sterilizing medical equipment. The active ingredient in acetic acid can effectively kill mycobacterium causing tuberculosis. Acetic acid might be used as a disinfectant to drug resistant TB. Effort for drug-resistant tuberculosis bacteria transmits serious biohazard risks and pulmonary disorders such as Chronic Obstructive Pulmonary Disease, Pulmonary Hypertension. Chlorine bleach is often used to disinfect TB cultures and clinical samples, but it is toxic. Diseases caused by mycobacteria, but non-TB mycobacteria are communal in the environment are resistant to disinfectants. When they contaminate the sites of surgery or cosmetic procedures, they cause serious allergies and infections.

Upon investigation the ability of non-TB mycobacteria was tested in research laboratories for resistance of disinfectants and antibiotics i.e., vinegar’s ability to kill mycobacteria. A procedure was carried out by acquaintance to 6% acetic acid, which is just slightly more concentrated than super market vinegar, for 30 minutes, reduces the no.of mycobacteria from around 100 million to undetectable levels. Mycobacterium abscessus, one of the most resistant and pathogenic of the non-TB mycobacteria was most effective against acetic acid.

Depending upon the prevalence rate there is a real need for a less toxic and less expensive disinfectants that can eliminate TB and non TB mycobacteria in today’s world especially in poor countries. Tuberculosis (TB) remains a major global cause of health problem. Identifying new drug targets and candidate drugs is a major priority to minimize the death rate caused due to tuberculosis. Efforts have been started worldwide to minimize tuberculosis and improvise the function of lung also to develop better-quality rifampin derivatives that are unaffected by alterations of the rifampin binding site on Mtb RNAP and that thus can eradicate rifampin-resistant TB bacteria.